CAS OpenIR  > 膜生物学国家重点实验室
The late increase in intracellular free radical oxygen species during apoptosis is associated with cytochrome c release, caspase activation, and mitochondrial dysfunction
Source PublicationCell Death and Differentiation
2003
Volume10Issue:3Pages:323-334
Chen Q; Chai YC; Mazumder S; Jiang C; Macklis R; Chisolm GM; Almasan A
Document Type期刊论文
Identifierhttp://ir.ioz.ac.cn/handle/000000/10393
Collection膜生物学国家重点实验室
Recommended Citation
GB/T 7714
Chen Q,Chai YC,Mazumder S,et al. The late increase in intracellular free radical oxygen species during apoptosis is associated with cytochrome c release, caspase activation, and mitochondrial dysfunction[J]. Cell Death and Differentiation,2003,10(3):323-334.
APA Chen Q.,Chai YC.,Mazumder S.,Jiang C.,Macklis R.,...&Almasan A.(2003).The late increase in intracellular free radical oxygen species during apoptosis is associated with cytochrome c release, caspase activation, and mitochondrial dysfunction.Cell Death and Differentiation,10(3),323-334.
MLA Chen Q,et al."The late increase in intracellular free radical oxygen species during apoptosis is associated with cytochrome c release, caspase activation, and mitochondrial dysfunction".Cell Death and Differentiation 10.3(2003):323-334.
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