Knowledge Management System of Institute of Zoology, CAS
| The late increase in intracellular free radical oxygen species during apoptosis is associated with cytochrome c release, caspase activation, and mitochondrial dysfunction | |
| Source Publication | Cell Death and Differentiation
 |
| 2003 | |
| Volume | 10Issue:3Pages:323-334 |
| Chen Q; Chai YC; Mazumder S; Jiang C; Macklis R; Chisolm GM; Almasan A | |
| Document Type | 期刊论文 |
| Identifier | http://ir.ioz.ac.cn/handle/000000/10393 |
| Collection | 膜生物学国家重点实验室 |
| Recommended Citation GB/T 7714 | Chen Q,Chai YC,Mazumder S,et al. The late increase in intracellular free radical oxygen species during apoptosis is associated with cytochrome c release, caspase activation, and mitochondrial dysfunction[J]. Cell Death and Differentiation,2003,10(3):323-334. |
| APA | Chen Q.,Chai YC.,Mazumder S.,Jiang C.,Macklis R.,...&Almasan A.(2003).The late increase in intracellular free radical oxygen species during apoptosis is associated with cytochrome c release, caspase activation, and mitochondrial dysfunction.Cell Death and Differentiation,10(3),323-334. |
| MLA | Chen Q,et al."The late increase in intracellular free radical oxygen species during apoptosis is associated with cytochrome c release, caspase activation, and mitochondrial dysfunction".Cell Death and Differentiation 10.3(2003):323-334. |
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| 149-The late increas（547KB） | 开放获取 | ODC PDDL | View Download | |||
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