The late increase in intracellular free radical oxygen species during apoptosis is associated with cytochrome c release, caspase activation, and mitochondrial dysfunction

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	The late increase in intracellular free radical oxygen species during apoptosis is associated with cytochrome c release, caspase activation, and mitochondrial dysfunction
发表期刊	Cell Death and Differentiation
	2003
卷号	10 期号:3 页码:323-334
	Chen Q; Chai YC; Mazumder S; Jiang C; Macklis R; Chisolm GM; Almasan A
文献类型	期刊论文
条目标识符	http://ir.ioz.ac.cn/handle/000000/10393
专题	膜生物学国家重点实验室
推荐引用方式 GB/T 7714	Chen Q,Chai YC,Mazumder S,et al. The late increase in intracellular free radical oxygen species during apoptosis is associated with cytochrome c release, caspase activation, and mitochondrial dysfunction[J]. Cell Death and Differentiation,2003,10(3):323-334.
APA	Chen Q.,Chai YC.,Mazumder S.,Jiang C.,Macklis R.,...&Almasan A.(2003).The late increase in intracellular free radical oxygen species during apoptosis is associated with cytochrome c release, caspase activation, and mitochondrial dysfunction.Cell Death and Differentiation,10(3),323-334.
MLA	Chen Q,et al."The late increase in intracellular free radical oxygen species during apoptosis is associated with cytochrome c release, caspase activation, and mitochondrial dysfunction".Cell Death and Differentiation 10.3(2003):323-334.