H2A.Z.1 crosstalk with H3K56-acetylation controls gliogenesis through the transcription of folate receptor

	H2A.Z.1 crosstalk with H3K56-acetylation controls gliogenesis through the transcription of folate receptor
Source Publication	NUCLEIC ACIDS RESEARCH
	2018
Volume	46 Issue:17 Pages:8817-8831
	Libo Su; Wenlong Xia; Tianjin Shen; Qingli Liang; Wenwen Wang; Li H(李红); Jiao JW(焦建伟)
Document Type	期刊论文
Identifier	http://ir.ioz.ac.cn/handle/000000/13400
Collection	干细胞与生殖生物学国家重点实验室
Recommended Citation GB/T 7714	Libo Su,Wenlong Xia,Tianjin Shen,et al. H2A.Z.1 crosstalk with H3K56-acetylation controls gliogenesis through the transcription of folate receptor[J]. NUCLEIC ACIDS RESEARCH,2018,46(17):8817-8831.
APA	Libo Su.,Wenlong Xia.,Tianjin Shen.,Qingli Liang.,Wenwen Wang.,...&焦建伟.(2018).H2A.Z.1 crosstalk with H3K56-acetylation controls gliogenesis through the transcription of folate receptor.NUCLEIC ACIDS RESEARCH,46(17),8817-8831.
MLA	Libo Su,et al."H2A.Z.1 crosstalk with H3K56-acetylation controls gliogenesis through the transcription of folate receptor".NUCLEIC ACIDS RESEARCH 46.17(2018):8817-8831.

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