Knowledge Management System of Institute of Zoology, CAS
H2A.Z.1 crosstalk with H3K56-acetylation controls gliogenesis through the transcription of folate receptor | |
Source Publication | NUCLEIC ACIDS RESEARCH
![]() |
2018 | |
Volume | 46Issue:17Pages:8817-8831 |
Libo Su; Wenlong Xia; Tianjin Shen; Qingli Liang; Wenwen Wang; Li H(李红); Jiao JW(焦建伟) | |
Document Type | 期刊论文 |
Identifier | http://ir.ioz.ac.cn/handle/000000/13400 |
Collection | 干细胞与生殖生物学国家重点实验室 |
Recommended Citation GB/T 7714 | Libo Su,Wenlong Xia,Tianjin Shen,et al. H2A.Z.1 crosstalk with H3K56-acetylation controls gliogenesis through the transcription of folate receptor[J]. NUCLEIC ACIDS RESEARCH,2018,46(17):8817-8831. |
APA | Libo Su.,Wenlong Xia.,Tianjin Shen.,Qingli Liang.,Wenwen Wang.,...&焦建伟.(2018).H2A.Z.1 crosstalk with H3K56-acetylation controls gliogenesis through the transcription of folate receptor.NUCLEIC ACIDS RESEARCH,46(17),8817-8831. |
MLA | Libo Su,et al."H2A.Z.1 crosstalk with H3K56-acetylation controls gliogenesis through the transcription of folate receptor".NUCLEIC ACIDS RESEARCH 46.17(2018):8817-8831. |
Files in This Item: | Download All | |||||
File Name/Size | DocType | Version | Access | License | ||
H2A.Z.1 crosstalk wi(10867KB) | 期刊论文 | 出版稿 | 开放获取 | CC BY-NC-SA | View Download |
Items in the repository are protected by copyright, with all rights reserved, unless otherwise indicated.
Edit Comment