CAS OpenIR  > 干细胞与生殖生物学国家重点实验室
H2A.Z.1 crosstalk with H3K56-acetylation controls gliogenesis through the transcription of folate receptor
Source PublicationNUCLEIC ACIDS RESEARCH
2018
Volume46Issue:17Pages:8817-8831
Libo Su; Wenlong Xia; Tianjin Shen; Qingli Liang; Wenwen Wang; Li H(李红); Jiao JW(焦建伟)
Document Type期刊论文
Identifierhttp://ir.ioz.ac.cn/handle/000000/13400
Collection干细胞与生殖生物学国家重点实验室
Recommended Citation
GB/T 7714
Libo Su,Wenlong Xia,Tianjin Shen,et al. H2A.Z.1 crosstalk with H3K56-acetylation controls gliogenesis through the transcription of folate receptor[J]. NUCLEIC ACIDS RESEARCH,2018,46(17):8817-8831.
APA Libo Su.,Wenlong Xia.,Tianjin Shen.,Qingli Liang.,Wenwen Wang.,...&焦建伟.(2018).H2A.Z.1 crosstalk with H3K56-acetylation controls gliogenesis through the transcription of folate receptor.NUCLEIC ACIDS RESEARCH,46(17),8817-8831.
MLA Libo Su,et al."H2A.Z.1 crosstalk with H3K56-acetylation controls gliogenesis through the transcription of folate receptor".NUCLEIC ACIDS RESEARCH 46.17(2018):8817-8831.
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